Peter Daszak and Linfa Wang, authors on DEFUSE proposal both point to Baric as having inserted the furin cleavage site that gave SCV2 pandemic potential.
Baric made the virus for Moderna who is his close collaborator. Fauci is covering this up because NIH co-owns the vaccine with Moderna.
theintercept.com
Other urls found in this thread:
elifesciences.org
rupress.org
ncbi.nlm.nih.gov
documentcloud.org
youtube.com
youtube.com
twitter.com
ncbi.nlm.nih.gov
youtube.com
twitter.com
Bump
Who own the patents of the sequences found on the virus?
The Moderna patent sequence was inserted to kill people off with cancer. It doesn't explain the furin cleavage site however.
The UNC was studying the RRAR cleavage site in human airway epithelium in 2018. Which appears as the FCS of SCV2.
elifesciences.org
rupress.org
Good thread OP. Repost if archived early. There are anons here that would want to see this
Fortuitous rolls, OP.
How does the "Moderna patent sequence" kill off those with cancer, though?
>Scientists disagree over an issue where there’s no definitive proof. And for this issue, there’s no definitive proof. And there may never be.
first it was impossible. a conspiracy theory. now we're here. what's next?
>we couldn't have known the virus escaped due to Chynas bad safety protocols
lol
NEVER TOOK THE CLOTSHOT
NEVER WILL
DIE SHEEP BASTARDS
ncbi.nlm.nih.gov
This is how.
Back when Obama paused GOF, he gave the CIA (USAID-EcoHealth) the opportunity to move the research to Wuhan. This was by design, it was leaked in China next to a lab that was doing similar work in order to cover their tracks.
He took the jab tough!
What do you mean he's larping?
You can clearly see the needle and the vials:
youtube.com
>If you ever want to be prepare to make money during the next pandemic
youtube.com
The progenitor genome The root of the mutation tree is the most recent common ancestor (MRCA) of all the genomes analyzed, which gave rise to two early coronavirus lineages (ν and α; Fig. 1). The MRCA genome was the progenitor of all SARS-CoV-2 infections globally, henceforth proCoV2, and was likely carried by the first case of human transmission in the COVID-19 pandemic (index case)20. It existed on or before December 24, 2019, a date for which we have the sequence of SARS-CoV-2 infection in Wuhan, China (Wuhan-1; EPI_ISL 402123). A comparison of proCoV2 with Wuhan-1 genomes revealed three differences in the 49 positions, which was also true for other reference genomes (Fig. 2c). This suggests that the Wuhan-1 and the other earliest sampled genomes are derived coronavirus strains that arose from proCoV2 after the divergence of ν and α lineages (Fig. 1). The Wuhan-1 strain evolved by three successive α mutations in the progenitor (α1, α2, and α3), a progression that is statistically supported (BCL = 100%). This high resolution is made possible by 896 intermediate genomes containing one or two α variants in the 29KG dataset. Importantly, three closely-related non-human coronavirus genomes (bats and pangolin) all have the same base at these positions as does the proCoV2 genome, suggesting that the ancestral genome did not contain α variants. Furthermore, genomes with ν variants of proCoV2 do not contain the other 47 variants, all of which occurred on the genomes containing α1-α3 that supports the inference that coronaviruses lacking α variants were the ancestors of Wuhan-1 and other genomes sampled in December 2019 in China (Fig. 2c). Therefore, we conclude that Wuhan-1 was not the direct ancestor of all the coronavirus infections globally.
More Baric's quotes:
twitter.com
Did proCoV2 propagate in the human population in 2020? A comparison of the proCoV2 genetic fingerprint (49 positions) in the 29KG collection revealed three matches in China (Fujian, Guangdong, and Hangzhou) and three in the US (Washington) in January 2020 (Fig. 2c). One more match was found in New York in March 2020, and the ν mutant of proCoV2 was first sampled 59 days after the Wuhan-1 strain. This means that the progenitor coronavirus spread and mutated in the human population for weeks and months after the first reported COVID-19 cases.
Because proCoV2 is three bases different from the Wuhan-1 genome sampled on December 24, 2019, we estimate that the divergence of earliest variants of proCoV2 occurred 5.8 – 8.1 weeks prior based on the range of possible mutation rates of coronavirus genomes20. This timeline puts the presence of proCoV2 late-October to mid-November 2019 that is consistent with some other reports, including the report of a fragment of spike protein identical to Wuhan-1 in early December in Italy18,20,29–31.
Ralph Baric at the Gain of Function Symposium
Session 5: Potential Benefits of GOF Research II: Treatment and Response
youtube.com
ill have what hes having
nothing....
